Hormones

17beta-estradiol suppresses TLR3-induced cytokine and chemokine production

Published in Proteomics Weekly, March 20th, 2006

17beta-estradiol (E2) can markedly alter the innate immune response to dsRNA, providing a previously unreported process by which E2 can alter immune responses.

According to recent research from the United States, "The human endometrium is an important site for contact between the host and pathogens ascending the reproductive tract, and thus plays an important role in female reproductive tract immunity. Previous work in our laboratory has suggested that Toll-like receptors (TLRs) are involved in endometrial epithelial recognition of pathogens and that ligation of endometrial TLRs results in the production of cytokines and chemokines important for both immune and...

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Source: Proteomics Weekly (2006-03-20)

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