Pathogenesis (TB)
"Loss of oxyR in M. tuberculosis."
Published in TB and Outbreaks Week, June 14th, 1999
NIH Grant Number: 1R01AI42999-01. Grant Expires: March 31, 2003. Principle Investigator: Vojo Deretic. Performing Organization: University of Michigan, Ann Arbor, Michigan 48109. Description: "M. tuberculosis is a natural mutant in the putative central regulator of oxidative stress response, oxyR. All M. tuberculosis strains investigated to date have nearly identical lesions in oxyR, and the elimination of oxyR function may have coincided with or directly participated in the evolution of M. tuberculosis into the potent contemporary human pathogen. The loss of oxyR has a dual significance for M. tuberculosis...
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