Helicobacter Pylori

Transient apoptosis of lymphocytes following infection may be homeostatic

Published in TB and Outbreaks Week, August 5th, 2003

In the rhesus macaque model, Helicobacter pylori infection is followed by transient apoptosis of gastric lymphocytes that may be part of normal homeostastis after T cell activation.

"In vitro studies suggest that H. pylori induces apoptosis in gastric epithelial cells and perhaps in gastric lymphocytes as well. However, the early effects of H. pylori infection on lymphocyte apoptosis have not been examined in experimental animal models, nor have studies been performed using markers specific for T cells and T-cell subsets," scientists in the United States report.

"Gastric T-cell apoptosis and Fas ligand expression were examined by...

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Source: TB and Outbreaks Week (2003-08-05)

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