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Tuberculosis
Research conducted at Tokushima Bunri University has updated our knowledge about tuberculosis
June 15th, 2009
According to recent research published in the journal Anticancer Research, "Mycobacterium tuberculosis (Mtb), an intracellular pathogen, is phagocytosed by alveolar macrophage but it is not digested; it survives, proliferates and establishes Mtb infections. The long-term survival mechanism of Mtb is not yet clear." "The host's immune response to Mtb is mainly mediated by, a Toll-like receptor 2 (TLR2) in macrophages. In the earl), stage of the immune response by macrophage activation through TLR2, the proliferation of Mtb is suppressed and there is a direct bactericidal effect or induction of apoptosis in infected macrophages. This indicates that TLR2 signaling...
Source: Tuberculosis Week (2009-06-15)
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