Herpesvirus Vaccine

CD4 T cells critical in vaccination against gamma herpesvirus latency

Published in Vaccine Weekly, August 25th, 2004

CD4 T cells play a critical role in an antibody-independent mechanism of vaccination against gamma herpesvirus latency.

"We have previously demonstrated that it is possible to effectively vaccinate against long-term murine gamma herpesvirus 68 (gammaHV68) latency by using a reactivation-deficient virus as a vaccine. Immune antibody was capable of recapitulating aspects of this vaccination. This led us to determine whether antibody is required for vaccination against latency. Using mice lacking antigen-specific antibody responses, we demonstrate here that antibody and B cells are not required for vaccination against latency," investigators in the United States...

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Source: Vaccine Weekly (2004-08-25)

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