Published in Vaccine Weekly, May 24th, 2006
"Virus-encoded modulation of apoptosis may serve as a mechanism to enhance cell survival and virus persistence. The impact of productive varicella-zoster virus (VZV) infection on apoptosis appears to be cell type-specific, as infected human sensory neurons are resistant to apoptosis, yet human fibroblasts readily become apoptotic. We sought to identify the viral gene product responsible for this anti-apoptotic phenotype in primary human sensory neurons," scientists in Australia and the...
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