Published in Virus Weekly, April 29th, 2003
The majority (80%) of patients newly infected with the hepatitis C virus (HCV) will go on to develop chronic infection. This led J.G. Bode and colleagues to hypothesize that "because SOCS molecules have been recognized to be powerful inhibitors of cytokine signaling via the Jak/STAT pathway, virus-induced expression of these molecules should be an efficient instrument to counteract the cellular response toward interferons (IFNs), an essential part of first...
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