Reproductive Immunology
Fetal and maternal TGF-beta 1 may combine to maintain pregnancy in mice
Published in Women's Health Weekly, July 29th, 2004
According to researchers at the University of Otago in New Zealand, "One of the mysteries of pregnancy is why a mother does not reject her fetuses. Cytokine-modulation of maternal-fetal interactions is likely to be important. However, mice deficient in transforming growth factor-beta1 (TGFbeta1) and other cytokines are able to breed, bringing this hypothesis into question.
"The phenotype of TGFbeta1 null-mutant mice varies with genetic background," said I.S. McLennan and colleagues.
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